Oligodendrocyte damage and subsequent axonal demyelination is a hallmark of this disease.
少突膠質(zhì)細(xì)胞損害,繼而軸突脫髓鞘為本病特征.
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Conclusion EAE is characterised by perivascular inflammatory cell infiltration and demyelination in white matter.
結(jié)論EAE的病理改變主要為血管周圍炎性浸潤及白質(zhì)脫髓鞘.
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Significant endoneurial edema, inflammation reaction and demyelination can be seen in the DRGs and NRs.
自體髓核移植可致脊神經(jīng)節(jié)、神經(jīng)根明顯的水腫 、 炎性反應(yīng)、脫髓鞘等病理改變.
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